STRAIGHT-IN enables high-throughput targeting of large DNA payloads in human pluripotent stem cells

Albert Blanch-Asensio, Catarina Grandela, Karina O. Brandão, Tessa de Korte, Hailiang Mei, Yavuz Ariyurek, Loukia Yiangou, Mervyn P.H. Mol, Berend J. van Meer, Susan L. Kloet, Christine L. Mummery, Richard P. Davis*

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

24 Citations (Scopus)
150 Downloads (Pure)

Abstract

Inserting large DNA payloads (>10 kb) into specific genomic sites of mammalian cells remains challenging. Applications ranging from synthetic biology to evaluating the pathogenicity of disease-associated variants for precision medicine initiatives would greatly benefit from tools that facilitate this process. Here, we merge the strengths of different classes of site-specific recombinases and combine these with CRISPR-Cas9-mediated homologous recombination to develop a strategy for stringent site-specific replacement of genomic fragments at least 50 kb in size in human induced pluripotent stem cells (hiPSCs). We demonstrate the versatility of STRAIGHT-IN (serine and tyrosine recombinase-assisted integration of genes for high-throughput investigation) by (1) inserting various combinations of fluorescent reporters into hiPSCs to assess the excitation-contraction coupling cascade in derivative cardiomyocytes and (2) simultaneously targeting multiple variants associated with inherited cardiac arrhythmic disorders into a pool of hiPSCs. STRAIGHT-IN offers a precise approach to generate genetically matched panels of hiPSC lines efficiently and cost effectively.

Original languageEnglish
Article number100300
Number of pages23
JournalCell Reports Methods
Volume2
Issue number10
Early online date22 Sept 2022
DOIs
Publication statusPublished - 24 Oct 2022

Keywords

  • Bxb1 integrase
  • cardiomyocyte
  • Cre recombinase
  • CRISPR-Cas9
  • disease modeling
  • human pluripotent stem cells
  • site-specific recombination
  • synthetic gene circuit
  • targeted gene modification

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